Search results for "substance p"

showing 10 items of 64 documents

Non-Rapid Eye Movement Sleep Parasomnias and Migraine: A Role of Orexinergic Projections

2018

Introduction: Sleep and migraine share a common pathophysiological substrate, although the underlying mechanisms are unknown. The serotonergic and orexinergic systems are both involved in the regulation of sleep/wake cycle, and numerous studies show that both are involved in the migraine etiopathogenesis. These two systems are anatomically and functionally interconnected. Our hypothesis is that in migraine a dysfunction of orexinergic projections on the median raphe (MR) nuclei, interfering with serotonergic regulation, may cause Non-Rapid Eye Movement parasomnias, such as somnambulism. Hypothesis/theory: Acting on the serotonergic neurons of the raphe nuclei, the dysfunction of orexinergic…

0301 basic medicineSerotonergic systemMigraine; Orexinergic system; Pro-inflammatory peptides; Serotonergic system; Sleep-wake rhythm; Neurology; Neurology (clinical)Substance PCalcitonin gene-related peptidePro-inflammatory peptideSerotonergicNon-rapid eye movement sleeplcsh:RC346-429sleep–wake rhythmMigraine; Orexinergic system; Pro-inflammatory peptides; Serotonergic system; Sleep-wake rhythm;Settore M-PSI/04 - Psicologia Dello Sviluppo E Psicologia Dell'Educazione03 medical and health sciencesTrigeminal ganglionchemistry.chemical_compound0302 clinical medicinePro-inflammatory peptidesSleep-wake rhythmHypothesis and TheoryMedicinelcsh:Neurology. Diseases of the nervous systemMigraineMigraine; Orexinergic system; Pro-inflammatory peptides; Serotonergic system; Sleep-wake rhythmbusiness.industryOrexinergic systemserotonergic system orexinergic system sleep–wake rhythm migraine pro-inflammatory peptidesSettore MED/39 - Neuropsichiatria InfantileOrexin030104 developmental biologyNeurologychemistryNeurology (clinical)SerotoninbusinessRaphe nucleiNeuroscience030217 neurology & neurosurgeryNeuroscienceFrontiers in Neurology
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Interaction of calcitonin gene related peptide (CGRP) and substance P (SP) in human skin.

2016

Calcitonin gene related peptide (CGRP) and substance P (SP) are neuropeptides that are simultaneously released from nociceptive C-fibers. CGRP is a potent vasodilator, inducing a long-lasting increase in superficial skin blood flow, whereas SP induces only a brief vasodilation but a significant plasma extravasation. CGRP and SP may play important roles in the pathophysiology of various pain states but little is known about their interaction. Different concentrations of SP (ranging from 10-5M to 10-9M) were applied to the volar forearm of 24 healthy subjects via dermal microdialysis. SP was applied either alone or in combination with CGRP10-9M and CGRP 10-6M. As expected, SP induced a transi…

0301 basic medicinemedicine.medical_specialtyMicrodialysisCalcitonin Gene-Related PeptideVasodilationSubstance PCalcitonin gene-related peptideSubstance P03 medical and health sciencesCellular and Molecular Neurosciencechemistry.chemical_compoundYoung Adult0302 clinical medicineEndocrinologyInternal medicinemedicineHumansNeprilysinSkinReceptor activity-modifying proteinintegumentary systemDose-Response Relationship DrugEndocrine and Autonomic SystemsChemistryGeneral MedicineCALCRLExtravasationVasodilationForearm030104 developmental biologyEndocrinologyNeurologyRegional Blood Flow030217 neurology & neurosurgeryNeuropeptides
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Binding of basic amphipathic peptides to neutral phospholipid membranes: a thermodynamic study applied to dansyl-labeled melittin and substance P ana…

1997

A thermodynamic approach is proposed to quantitatively analyze the binding isotherms of peptides to model membranes as a function of one adjustable parameter, the actual peptide charge in solution z(p)+. The main features of this approach are a theoretical expression for the partition coefficient calculated from the molar free energies of the peptide in the aqueous and lipid phases, an equation proposed by S. Stankowski [(1991) Biophysical Journal, Vol. 60, p. 341] to evaluate the activity coefficient of the peptide in the lipid phase, and the Debye-Huckel equation that quantifies the activity coefficient of the peptide in the aqueous phase. To assess the validity of this approach we have s…

Activity coefficientProtein ConformationLipid BilayersMolecular Sequence DataBiophysicsPhospholipidPeptideSubstance PBiochemistryMelittinBiomaterialschemistry.chemical_compoundElectrochemistryOrganic chemistryAmino Acid Sequencechemistry.chemical_classificationDansyl CompoundsAqueous solutionTransglutaminasesChemistryOrganic ChemistryGeneral MedicineMelittenPartition coefficientCrystallographyMembraneSpectrometry FluorescenceIonic strengthThermodynamicsBiopolymers
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Binding of a fluorescent dansylcadaverine-substance P analogue to negatively charged phospholipid membranes.

2000

Abstract We have investigated the binding of a new dansylcadaverine derivative of substance P (DNC-SP) with negatively charged small unilamellar vesicles composed of a mixture of phosphatidylcholine (PC) and either phosphatidylglycerol (PG) or phosphatidylserine (PS) using fluorescence spectroscopic techniques. The changes in fluorescence properties were used to obtain association isotherms at variable membrane negative charges and at different ionic strengths. The experimental association isotherms were analyzed using two binding approaches: (i) the Langmuir adsorption isotherm and the partition equilibrium model, that neglect the activity coefficients; and (ii) the partition equilibrium m…

Activity coefficientVesicleLipid BilayersAnalytical chemistryLangmuir adsorption modelCharge densityGeneral MedicineSubstance PBiochemistryBinding constantFluorescencechemistry.chemical_compoundsymbols.namesakeMembranechemistryModels ChemicalStructural BiologyPhosphatidylcholinePartition equilibriumCadaverinesymbolsMolecular BiologyPhospholipidsFluorescent DyesProtein BindingInternational journal of biological macromolecules
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Inhibition of neuropeptide degradation suppresses sweating but increases the area of the axon reflex flare.

2013

The neuropeptides CGRP (calcitonin gene-elated peptide) and substance P (SP) mediate neurogenic inflammation. Both are degraded by the neutral endopeptidase (NEP) which can be blocked by phosphoramidon. The aim was to evaluate the effect of NEP inhibition on sweating and vasodilatation. Dermal microdialysis was performed on the skin of 39 subjects. Two fibres were perfused with phosphoramidon (0.01%, 0.02% or 0.2%), two with saline. Acetylcholine (ACh) was either added to the microdialysis perfusate (n = 30, 10(-2)  m) or thermoregulatory sweating was induced (n = 9). Co-application of phosphoramidon reduced cholinergic and thermoregulatory sweating. However, the flare size - a localized in…

AdultMaleMicrodialysismedicine.medical_specialtyCalcitonin Gene-Related PeptideNeuropeptideSubstance PSweatingDermatologyCalcitonin gene-related peptideSubstance PBiochemistrychemistry.chemical_compoundInternal medicineReflexmedicineHumansProtease InhibitorsMolecular BiologySkinNeurogenic inflammationintegumentary systemChemistryPhosphoramidonGlycopeptidesrespiratory systemAxonsEndocrinologyCholinergicFemaleNeprilysinAcetylcholinemedicine.drugBody Temperature RegulationExperimental dermatology
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Serum levels of substance P are decreased in patients with type 1 diabetes.

2000

Morphological and immunohistochemical studies in diabetic subjects have shown a depletion of the neuropeptide substance P (SP) in the central and peripheral nervous system. This is the first study investigating serum levels of substance P in type 1 diabetes patients (n=50) and controls (n=75) by means of an enzyme immunoassay. The serum level of SP was significantly decreased in the diabetic group compared to the control group (10.12+/-0.29 vs. 12.25+/-0.38 pg/ml; p<0.0001). In diabetic patients, there was no correlation of substance P levels with age, serum creatinine, albuminuria, total cholesterol, HDL- or LDL-cholesterol, triglycerides, HbA1c, type or duration of diabetes and gender. Fu…

AdultMalemedicine.medical_specialtyDiabetic neuropathyAdolescentEndocrinology Diabetes and MetabolismSubstance PSubstance PImmunoenzyme Techniqueschemistry.chemical_compoundEndocrinologyReference ValuesInternal medicineDiabetes mellitusBlood plasmaInternal MedicinemedicineHumansAgedType 1 diabetesCreatininebusiness.industryGeneral MedicineMiddle Agedmedicine.diseasePeripheral neuropathyEndocrinologyCholesterolDiabetes Mellitus Type 1chemistryAlbuminuriaFemalemedicine.symptombusinessExperimental and clinical endocrinologydiabetes : official journal, German Society of Endocrinology [and] German Diabetes Association
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Lack of genetic association of neutral endopeptidase (NEP) with complex regional pain syndrome (CRPS)

2010

Complex regional pain syndrome (CRPS) is a condition that is characterized by severe pain and exaggerated neurogenic inflammation, which may develop after injury or surgery. Neurogenic inflammation is mediated by neuropeptides, such as calcitonin gene-related peptide (CGRP) and substance P (SP) that are released from nociceptors. Genetic factors may play a role in CRPS as was suggested by the occurrence of familial cases and several genetic association studies investigating mainly the human leukocyte antigen (HLA) system. Here we investigated the role of neutral endopeptidase (NEP), a key enzyme in neuropeptide catabolism. NEP dysfunction resulting in reduced inactivation of neuropeptides m…

AdultMalemedicine.medical_specialtyLinkage disequilibrium5' Flanking RegionSubstance PHuman leukocyte antigenBiologyCalcitonin gene-related peptideLinkage Disequilibriumchemistry.chemical_compoundInternal medicinemedicineHumansGenetic Predisposition to DiseaseDinucleotide RepeatsPromoter Regions GeneticNeprilysinGenetic Association StudiesGenetic associationNeurogenic inflammationPolymorphism GeneticGeneral NeurosciencefungiMiddle Agedmedicine.diseaseCRPS Pain NEP Association reflex sympathetic dystrophy syndrome type-i facilitated neurogenic inflammation nociceptive abnormalities alzheimers-disease neprilysin gene rat model enkephalinase prevalence dystoniaEndocrinologyComplex regional pain syndromechemistryCase-Control StudiesFemaleNeprilysinComplex Regional Pain Syndromes
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Substance-P-induced protein extravasation is bilaterally increased in complex regional pain syndrome.

2003

Pain, mechanical hyperalgesia, edema, increased skin temperature, and skin reddening are characteristic symptoms of acute complex regional pain syndrome (CRPS). We have recently demonstrated facilitated neurogenic inflammation on the affected limb. To further elucidate the underlying mechanisms, exogenous substance P (SP) in ascending concentrations (10(-9), 10(-8), 10(-7), 10(-6) M) was intradermally applied to the affected and the unaffected limbs, respectively, in two groups of 11 CRPS patients each using the microdialysis technique. Fourteen healthy volunteers served as controls for SP application, and 9 volunteers and 10 patients served as controls for saline perfusion. Dialysate prote…

AdultMalemedicine.medical_specialtyMicrodialysisInjections Intradermalmedicine.medical_treatmentMicrodialysisSubstance PSubstance Pchemistry.chemical_compoundDevelopmental NeuroscienceReference ValuesInternal medicineEdemamedicineHumansSalineSkinNeurogenic inflammationLegbusiness.industryBlood ProteinsMiddle Agedmedicine.diseaseExtravasationVasodilationComplex regional pain syndromeEndocrinologyNeurologychemistryRegional Blood FlowAnesthesiaHyperalgesiaArmFemalemedicine.symptomNeurogenic InflammationbusinessComplex Regional Pain SyndromesExperimental neurology
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Cizolirtine Citrate, an Effective Treatment for Symptomatic Patients with Urinary Incontinence Secondary to Overactive Bladder: A Pilot Dose-Finding …

2009

Abstract Background A dose-finding study was performed as the first step in the clinical development of the new drug, cizolirtine citrate. Objective To assess the efficacy and safety of cizolirtine citrate in overactive bladder with urinary incontinence. Design, setting, and participants Seventy-nine outpatients with clinical overactive bladder and/or urodynamic diagnosis of detrusor overactivity were randomized in a multicentre, 12-wk, double-blind, pilot trial. Interventions Patients received cizolirtine citrate 400mg bid (C400), cizolirtine citrate 200mg bid (C200), or placebo. Measurements Patients recorded efficacy variables in 7- and 14-d bladder diaries: urinary incontinence episodes…

Adultmedicine.medical_specialtyVomitingCalcitonin Gene-Related PeptideUrologyUrinary systemVision DisordersUrologyPilot ProjectsUrinary incontinenceSubstance PPlaceboDizzinesslaw.inventionYoung AdultDouble-Blind MethodRandomized controlled triallawmedicineHumansAdverse effectAgedAged 80 and overUrinary bladderDose-Response Relationship DrugUrinary Bladder Overactivebusiness.industryNauseaMiddle Agedmedicine.diseaseUrodynamicsUrinary Incontinencemedicine.anatomical_structureTolerabilityOveractive bladderPyrazolesFemalemedicine.symptombusinessEuropean Urology
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Tachykinin NK(2) receptors facilitate acetylcholine release from guinea-pig isolated trachea.

2000

The release of newly synthesised [3H]acetylcholine was evoked by electrical field stimulation (5 Hz, 600 pulses) of epithelium-deprived guinea-pig trachea strips after sensory neuropeptides depletion with 3 microM capsaicin. The selective tachykinin NK(2) receptor agonist [betaAla(8)]neurokinin A-(4-10) increased in a concentration-dependent manner the electrically-induced release of [3H]acetylcholine. The facilitatory effect was antagonised by the selective non-peptide tachykinin NK(2) receptor antagonist, SR 48968 (apparent pK(B) 8.9). The tachykinin NK(1) and NK(3) receptor agonists substance P methyl ester and senktide (both 10 and 100 nM), respectively, did not affect the evoked releas…

AgonistMalemedicine.medical_specialtymedicine.drug_classNeurokinin AGuinea PigsSubstance PIn Vitro TechniquesCholinechemistry.chemical_compoundPiperidinesInternal medicinemedicineAnimalsReceptorPharmacologyNeuronsReceptors Neurokinin-2Receptor antagonistAcetylcholineElectric StimulationPeptide FragmentsTracheaEndocrinologychemistryCapsaicinBenzamidesNeurokinin ACapsaicinTachykinin receptorAcetylcholinemedicine.drugEuropean journal of pharmacology
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